(+) Increase Font Size | (-) Decrease Font Size

Glaucoma Care at Berks Eye

As a referral center for the evaluation and treatment of glaucoma, Berks Eye Physicians & Surgeons brings to you the most advanced, proven methods of glaucoma diagnosis as well as comprehensive treatment options. As a fellowship-trained glaucoma specialist and surgeon with over 20 years of clinical experience, Francisco L. Tellez, M.D. will provide the glaucoma patient with individualized care plans and personalized one-on-one attention. Whether the care plan calls for medication, surgery, or simply monitoring exams, Dr. Tellez and his friendly, attentive staff will focus on you as a person and will offer the best treatment choices tailored to your individual needs. Our goal is to protect your vision and quality of life, and to care for you in the warm and pleasant environment that distinguishes Berks Eye.

What Is Glaucoma?

GLAUCOMA is a group of eye disorders that share the common feature of optic nerve damage. This damage is usually, but not always, caused by elevated fluid pressure within the eye. When pressure inside the eye increases, the optic nerve loses nerve fibers and blind spots can develop, resulting in loss of peripheral or even central vision. The higher the pressure, the greater the chance of damage to the optic nerve. Glaucoma is a leading cause of irreversible visual impairment and blindness in the United States and worldwide.

(Glaucoma Introduction)

What Is The Optic Nerve?

The OPTIC NERVE is the “cable” that exits from the back of the eye and carries the images we see to the brain. It is made up of millions of nerve fibers that start in the retina and end up in the visual processing centers of the back of the brain (the occipital cortex). A healthy optic nerve is necessary for good vision. When the fibers of the optic nerve are damaged in glaucoma, loss of the side vision can develop, which can progress to blindness.

(Optic Nerve)


(Glaucoma Cause)

Who Is at Risk for Glaucoma?

As with many other diseases, certain individuals may be more at risk of developing glaucoma than others. During your glaucoma evaluation, your ophthalmologist will collect a good deal of information to help determine your glaucoma risk factor profile. Below are some of the major risk factors for glaucoma:

  • A family history of glaucoma (especially first-degree relatives)
  • African or Hispanic (especially Mexican) ancestry
  • Anyone over age 60 or African descent over age 40
  • High myopia (nearsightedness)
  • Past injuries to the eyes
  • High blood pressure
  • Elevated intraocular pressure
  • Central corneal thickness less than 0.5 mm

(Glaucoma Risk Factors)

What Causes Glaucoma?

Let´s learn a new word and its definition. AQUEOUS HUMOR is a clear, watery fluid inside the eye that nourishes the cornea, iris, lens, and maintains intraocular pressure (IOP). This liquid is not part of the tears on the outer surface of the eye. You can think of the flow of aqueous fluid as a sink with the faucet turned on all the time. If the "drainpipe" gets clogged, water collects in the sink and the pressure builds up. If the drainage area of the eye (DRAINAGE ANGLE) is blocked or does not work properly, the fluid pressure inside the eye may increase, which can damage the optic nerve. The result of this pressure increase as it pushes against the nerve is constriction (or loss of) the side vision.

(How Fluid Circulates in the Eye )

What Are the Most Common Types of Glaucoma?

GLAUCOMA is a broad term used to describe a number of different conditions, all of which can result in optic nerve damage. Most of these conditions involve elevation of the intraocular pressure, but not always. The most common types of glaucoma-related conditions include:

Ocular Hypertension (OHT). A condition where the intraocular pressure (IOP) is elevated but without any evidence of optic nerve damage or visual field loss. Patients with OHT will need regular monitoring for glaucoma, since IOP elevation is a major risk factor for glaucoma development.

(Ocular Hypertension )

Glaucoma Suspect. A situation where significant glaucoma risk factors are present, or where the findings of the clinical exam raise the suspicion of possible glaucoma (such as elevated IOP or questionable changes in the optic nerve. These patients require glaucoma surveillance exams at regular intervals.

Primary Open Angle Glaucoma (POAG). This is the most common type of glaucoma in the United States. POAG develops when the drainage system of the eye (the TRABECULAR MESHWORK) stops working properly. There is increased resistance to aqueous fluid drainage, resulting in a buildup of aqueous within the eye, which leads to a rise in intraocular pressure (IOP). The elevated IOP causes loss of optic nerve fibers. In POAG the pressure elevation may occur slowly over a long period of time. Typically there are no symptoms such as pain or blurred vision and the patient may thus be totally unaware that they have the condition. If the disease is undetected or untreated, however, optic nerve damage can occur with irreversible loss of vision. Most often when patients notice visual loss from POAG, the disease has likely reached an advanced stage, so it is very important to get checked early if you know you may be at risk (see risk factors above).

(Open Angle Glaucoma )

Primary Angle Closure Glaucoma Suspect (Narrow Angle). A situation where the drainage angle of the eye is anatomically narrow, with crowding between the peripheral iris and the trabecular meshwork. A narrow angle increases the risk of angle closure glaucoma because the crowding in the angle makes it more likely that the iris will stick to the trabecular meshwork, forming adhesions and physically obstructing the drainage system. A narrow angle tends to worsen with time, especially as cataracts develop.

Primary Angle Closure Glaucoma. An anatomic closure of the drainage system that develops when the iris blocks the trabecular meshwork, generally in eyes that have narrow angles or crowding of structures within the eye. The closure can develop gradually over time with a rise in IOP without symptoms (chronic angle closure glaucoma), or suddenly (acute angle closure glaucoma) with symptoms that include pain, headache, nausea, vomiting, a red eye, and blurred vision. Acute angle closure glaucoma is a medical emergency which poses a permanent threat to the vision; it requires immediate attention to relieve the very high pressure that can develop.

(Narrow Angle Glaucoma)

Pseudoexfoliation Glaucoma. A condition where a white, powdery material is produced inside the eye. The material deposits in the trabecular meshwork, obstructing drainage of aqueous and causing a rise in IOP. It is most common in Scandinavian populations and rates vary widely in other ethnic groups.

Pigmentary Glaucoma. A condition where pigment is spontaneously released from the back surface of the iris. The pigment deposits in various structures inside the eye, including the fluid drainage system. Blockage of the trabecular meshwork by this pigment leads to IOP elevation. Pigmentary glaucoma tends to affect younger individuals, especially males.

(Iris)

Steroid-induced Glaucoma. Steroid medications that may be used to treat other conditions can cause a rise in IOP which can lead to optic nerve damage if not detected. The greatest risk would occur from topically administered steroid eye drops that are used to treat a number of ocular inflammatory problems, but IOP elevation can also develop from other types of steroid use, such as dermatologic cortisone-type preparations, prednisone or other oral steroid pills, or even injectable cortisone shots. Patients with an underlying predisposition towards glaucoma are more likely to develop IOP elevation or steroid-induced glaucoma. Patients taking steroid medications on a chronic or long-term basis need regular glaucoma monitoring examinations.

Neovascular Glaucoma. Glaucoma that develops due to the growth of abnormal blood vessels within the eye that block the trabecular meshwork and the drainage angle. These blood vessels most often form as a result of poor blood flow to the eye in conditions such as diabetic retinopathy , clots in the retinal circulation (vein occlusions), or blockages in the carotid artery. The treatment of neovascular glaucoma requires both the management of the elevated IOP and the underlying circulatory problem.

(Diabetic Retinopathy Description)

Normal Tension Glaucoma. In this type of glaucoma, there is evidence of optic nerve damage and/or visual field loss, despite IOP readings that consistently may be normal or even low. Factors other than intraocular pressure are likely at play, like poor blood flow to the optic nerve, or perhaps an excess of oxidative stresses that damage the optic nerve even without IOP elevation. Normal tension glaucoma patients may have an increased incidence of migraine headaches, Raynaud's phenomenon, or low blood pressure, conditions that nay be associated with impaired vascular stability or blood flow regulation. The treatment of normal tension glaucoma focuses on IOP reduction and on preventing very low blood pressure that may hinder proper blood flow to the optic nerve.

(Glaucoma Description)

How Do You Protect Your Vision from Glaucoma?

Because the visual loss from glaucoma often occurs slowly over a long period of time, the patient may be totally unaware that there is a vision-threatening problem. Typically there is no pain or blurred vision. In fact, glaucoma is known as the “sneak thief of sight.”

For this reason, early detection and treatment by an ophthalmologist is the key to prevent your vision from being robbed.

How Is Glaucoma Detected?

A glaucoma eye examination by your ophthalmologist will include:

  • An assessment of your general health and medical history
  • A review of your glaucoma risk factors
  • A measurement of the visual acuity
  • A measurement of the intraocular pressure (TONOMETRY)
  • A measurement of the corneal thickness (PACHYMETRY) Click here to learn more about the Cornea.
  • An inspection of the drainage angle of the eye (GONIOSCOPY)
  • An evaluation of the optic nerve by direct visualization for any signs of damage (OPHTHALMOSCOPY)
  • An assessment of the visual field of each eye (PERIMETRY)


Optic Nerve Imaging

Newer, more sophisticated methods of optic nerve analysis using computerized digital imaging assist with the accuracy of the initial glaucoma diagnosis and provide a useful database of information for monitoring disease progression or stability.

Two such methods of optic nerve evaluation that utilize digital imaging technologies include CONFOCAL SCANNING LASER OPHTHALMOSCOPY (HRT) and OPTICAL COHERENCE TOMOGRAPHY (OCT).

HRT uses a scanning laser to create many parallel images of the optic nerve at increasing depth which are then reconstructed to create a 3-D image of the entire nerve head. These images are then stored in computer and are used for digital comparison with subsequent images. The HRT computer has special software to analyze the images and alert your ophthalmologist of changes in the structure of the optic nerve that may be due to glaucoma.

OCT uses special light beams that reflect off the retinal surface and are captured by the computer to create images of the optic nerve or the retinal layers around the optic nerve. The measurement of the retinal nerve fiber layer thickness around the optic nerve is of particular value in a glaucoma evaluation, since loss of nerve fibers will cause thinning of the nerve fiber layer.

How Is Glaucoma Treated?

Glaucoma cannot be cured, nor can lost vision be replaced or regenerated. However, further visual damage can be prevented by special glaucoma medications (eye drops or pills), laser surgery, or other microsurgical procedures.

Medications

A number of different glaucoma eye drops are available to lower the IOP. Most of the commonly used drops are dosed from one to three times per day. The drops work by either reducing fluid production inside the eye, or by increasing fluid outflow (drainage). Some of the drops also help to even out rises or fluctuations in IOP that can occur even at night, thereby leading to more level pressure control throughout the entire day. Eye drops can have side effects or interactions, so it is very important to inform your ophthalmologist of your medical conditions and of other medications you may be taking. Certain oral medications may also be used to treat glaucoma, although these are usually reserved for more severe cases or for short-term use.

Glaucoma Laser Surgery

Lasers have a number of applications in glaucoma treatment as described below. All laser procedures are brief, in-office or outpatient treatments.

Selective Laser Trabeculoplasty. A low power laser with a very short pulse of light, the SLT laser is applied directly to the trabecular meshwork to enhance or stimulate its normal drainage function. The laser causes a drop in IOP without cutting tissue or creating new holes or openings in the eye. SLT treatment offers many advantages to patients. It can be performed as an initial treatment instead of eye drops for patients who cannot tolerate medications or who desire fewer or no medications. It can be added to drops as a supplemental treatment for greater IOP lowering when necessary, allowing for less complex medical regimens. There are no systemic side effects. It is a safe, office-based procedure, performed simply with a drop of anesthetic in the eye. It is reimbursed by medical insurance. In some patients, the IOP lowering effect of the SLT laser may wear off over time, requiring a retreatment with the laser or a different therapeutic approach. SLT may be performed prior to cataract surgery in patients with coexisting glaucoma and cataract to maximize the chances of good IOP control with less need for glaucoma medications following cataract surgery.

Laser Iridotomy. In this procedure, a tiny hole is made in the peripheral iris to break an attack of acute angle closure glaucoma, or to prevent angle closure in patients with narrow anterior chamber angles (primary angle closure glaucoma suspects). Argon or YAG lasers are typically used for these treatments.

Laser Iridoplasty. An argon laser is used to slightly shrink the tissue of the mid-peripheral iris in order to pull it out of the drainage angle, to remove adhesions that may have already developed, or to prevent adhesions between the iris and trabecular meshwork that can lead to chronic angle closure glaucoma.

Endoscopic Cyclophotocoagulation (ECP). In this novel procedure, a tiny, delicate probe containing an argon laser delivery system is placed inside the eye. Through a fiberoptic line that is part of the probe, the surgeon can visualize the inside of the eye on a video camera. The laser light is applied to the ciliary body, the structure inside the eye that makes aqueous fluid. The laser energy causes a reduction in fluid production and a lowering of IOP. ECP is most commonly performed in conjunction with cataract surgery in patients that also have glaucoma.

(Ciliary Body).

Diode Cyclophotocoagulation (CPC). Laser energy is delivered to the ciliary body through an external approach with a special probe on the surface of the eye. This treatment is often reserved for advanced stages of glaucoma that may be difficult to control by other means, or for eyes with high pressure that are poor candidates for more conventional surgery.

Glaucoma Microsurgery

Different types of microsurgical interventions are available for IOP control and maintenance of vision, when medications or laser surgery do not provide sufficient control or are otherwise not appropriate.

Trabeculectomy. A procedure where a new drainage system is created with the patient´s own eye tissues. Aqueous fluid is rerouted from the blocked passageways to a new drainage site which results in lowering of the eye pressure. The standard glaucoma operation, trabeculectomy surgery has undergone refinements and modifications to improve the success rate, including the use of anti-scarring agents, new suture materials, and tissue handling techniques to enhance proper fluid outflow.

Aqueous Shunts. These are devices that are implanted in the eye to bypass the internal obstruction and to shunt the aqueous fluid to a new collecting system.

Schlemm´s Canal Surgery. Schlemm´s Canal is the channel that links the trabecular meshwork inside the eye with the venous collecting system on the surface of the eye, which is where the aqueous fluid eventually drains. For many years, the opening or tearing of this canal to facilitate the flow of aqueous out of the eye has been the mainstay of glaucoma surgery in congenital glaucoma (glaucoma in babies or small children). Similar techniques and concepts are now being evaluated and refined to treat adult glaucomas as well.

Cataract Surgery in the Glaucoma Patient

Patients with glaucoma often have coexisting cataracts.

(Cataract Description & Cataract Symptoms)

With the advances in modern, minimally invasive cataract surgery techniques that we enjoy today, including the use of small incisions and foldable implants, the majority of patients with controlled glaucoma can have successful cataract surgery alone, without special considerations for their glaucoma.

(Cataract Surgery Microincision Phaco)

In fact, recent studies show a real benefit of cataract surgery to improve overall glaucoma control. Cataract extraction alone will likely lower IOP in patients with high-pressure glaucoma. Cataract surgery is beneficial and often recommended for patients with significant narrow angles, because removing the cataract gets rid of the angle crowding and greatly reduces or eliminates the risk of angle closure.

Consultation with a glaucoma specialist can be of value in determining the best approach for cataract surgery when there is coexisting glaucoma. Pre-treatment with laser (SLT) prior to cataract surgery, for example, can provide better IOP control after the cataract operation, beyond the benefit that may be obtained from the cataract operation alone, with a greater reduction in the need for glaucoma drops. For glaucoma that is not well controlled or that is more advanced, cataract surgery can be combined with various glaucoma surgical techniques (trabeculectomy, aqueous shunts, ECP, etc) to ensure the best control of the glaucoma post-operatively and the best visual outcome long-term.

For further information on glaucoma, please visit the Glaucoma Research Foundation website at www.glaucoma.org